Skip to content

Hoxyl beads

Hybrid Quantum Chemistry magnetic beads

Menu
  • Home
  • miRNA
  • Isotype
  • PCR
  • peptide
  • Distributors
  • Polyclonal
  • Real-time
  • Recombinant
  • Contact Us
Menu
hoxyl

Szybka izolacja antygenów z komórek z adsorbentem gronkowcowym

Posted on 22 czerwca, 20214 maja, 2021 by Szymon

Izolacja ludzkiego genu, który hamuje zakażenie HIV-1 i jest tłumiony przez wirusowe białko Vif.

Viruses have developed various non-immune methods to counteract host-mediated mechanisms that confer resistance to an infection. The Vif (virion infectivity issue) proteins are encoded by primate immunodeficiency viruses, most notably human immunodeficiency virus-1 (HIV-1). These proteins are potent regulators of virus an infection and replication and are consequently important for pathogenic infections in vivo. HIV-1 Vif appears to be required in the course of the late levels of virus manufacturing for the suppression of an innate antiviral phenotype that resides in human T lymphocytes.

Thus, within the absence of Vif, expression of this phenotype renders progeny virions non-infectious. Right here, we describe a singular mobile gene, CEM15, whose transient or secure expression in cells that don’t usually categorical CEM15 recreates this phenotype, however whose antiviral motion is overcome by the presence of Vif. As a result of the Vif:CEM15 regulatory circuit is vital for HIV-1 replication, perturbing the circuit could also be a promising goal for future HIV/AIDS therapies.

hoxyl
hoxyl

VPREB3 ELISA KIT|Human

EF004206 Lifescience Market 96 Tests
EUR 826.8

TARGATT? Knock-in Mouse Cell Line Generation Kit (Master Cell Line)

AST-7001 Applied StemCell 1 Kit Ask for price
Description: 6 month

TARGATT? Knock-in iPSC Generation (Master Cell Line)

AST-1100 Applied StemCell 1 vial of 1X10^6 cells Ask for price
Description: 6 month

Human VPREB3 shRNA Plasmid

20-abx959234 Abbexa
  • EUR 961.20
  • EUR 1345.20
  • 150 µg
  • 300 µg

Vpreb3 sgRNA CRISPR Lentivector set (Rat)

K6154001 ABM 3 x 1.0 ug
EUR 406.8

Vpreb3 sgRNA CRISPR Lentivector set (Mouse)

K3123201 ABM 3 x 1.0 ug
EUR 406.8

T47D/GFP Cell Line

AKR-208 Cell Biolabs 1 vial
EUR 686.4
Description: T47D/GFP Cell Line stably expresses GFP and otherwise exhibits the same characteristics of the parental cell line.

VPREB3 sgRNA CRISPR/Cas9 All-in-One Lentivector set (Human)

K2617005 ABM 3 x 1.0 ug
EUR 451.2

TARGATT? Knock-in CHO Generation Kit (Master Cell Line)

AST-1200 Applied StemCell 1 Kit Ask for price
Description: 12 month

VPREB3 Recombinant Protein (Human)

RP034357 ABM 100 ug Ask for price

VPREB3 ORF Vector (Human) (pORF)

ORF011453 ABM 1.0 ug DNA
EUR 114

VPREB3 siRNA

20-abx939489 Abbexa
  • EUR 661.20
  • EUR 878.40
  • 15 nmol
  • 30 nmol

TARGATT? Knock-in HEK293 Generation Kit (Master Cell Line)

AST-1300 Applied StemCell 1 Kit Ask for price
Description: 12 month

Vpreb3 sgRNA CRISPR Lentivector (Rat) (Target 1)

K6154002 ABM 1.0 ug DNA
EUR 184.8

Vpreb3 sgRNA CRISPR Lentivector (Rat) (Target 2)

K6154003 ABM 1.0 ug DNA
EUR 184.8

Vpreb3 sgRNA CRISPR Lentivector (Rat) (Target 3)

K6154004 ABM 1.0 ug DNA
EUR 184.8

VPREB3 Peptide

42-296P ProSci 0.1 mg
EUR 405.6
Description: (IN) VPREB3 Peptide

VPREB3 Antibody

43594-100ul SAB 100ul
EUR 302.4

VPREB3 antibody

70R-21261 Fitzgerald 50 ul
EUR 522
Description: Rabbit polyclonal VPREB3 antibody

VPREB3 Antibody

DF15982 Affbiotech 100ul
EUR 420

VPREB3 Antibody

1-CSB-PA891724LA01HU Cusabio
  • EUR 380.40
  • EUR 402.00
  • 100ug
  • 50ug
Description: A polyclonal antibody against VPREB3. Recognizes VPREB3 from Human. This antibody is Unconjugated. Tested in the following application: ELISA

VPREB3 Antibody

1-CSB-PA025888GA01HU Cusabio
  • EUR 716.40
  • EUR 399.60
  • 150ul
  • 50ul
Description: A polyclonal antibody against VPREB3. Recognizes VPREB3 from Human. This antibody is Unconjugated. Tested in the following application: ELISA, WB

Vpreb3 sgRNA CRISPR Lentivector (Mouse) (Target 1)

K3123202 ABM 1.0 ug DNA
EUR 184.8

Vpreb3 sgRNA CRISPR Lentivector (Mouse) (Target 2)

K3123203 ABM 1.0 ug DNA
EUR 184.8

Vpreb3 sgRNA CRISPR Lentivector (Mouse) (Target 3)

K3123204 ABM 1.0 ug DNA
EUR 184.8

VPREB3 sgRNA CRISPR/Cas9 All-in-One Lentivector (Human) (Target 1)

K2617006 ABM 1.0 ug DNA
EUR 200.4

VPREB3 sgRNA CRISPR/Cas9 All-in-One Lentivector (Human) (Target 2)

K2617007 ABM 1.0 ug DNA
EUR 200.4

VPREB3 sgRNA CRISPR/Cas9 All-in-One Lentivector (Human) (Target 3)

K2617008 ABM 1.0 ug DNA
EUR 200.4

anti- VPREB3 antibody

FNab09425 FN Test 100µg
EUR 702
Description: Antibody raised against VPREB3

VPREB3 cloning plasmid

CSB-CL891724HU-10ug Cusabio 10ug
EUR 279.6
Description: A cloning plasmid for the VPREB3 gene.

293T Cell Lysate (Human embryonic kidney cell line)

LF-R0006 Abfrontier 200 ul
EUR 103.2
Description: 293T (Human embryonic kidney cell line) Whole Cell Lysate

VPREB3 Protein Vector (Human) (pPM-C-HA)

PV045811 ABM 500 ng
EUR 394.8

VPREB3 Protein Vector (Human) (pPB-C-His)

PV045809 ABM 500 ng
EUR 394.8

VPREB3 Protein Vector (Human) (pPB-N-His)

PV045810 ABM 500 ng
EUR 394.8

VPREB3 Protein Vector (Human) (pPM-C-His)

PV045812 ABM 500 ng
EUR 394.8

VPREB3 Conjugated Antibody

C43594 SAB 100ul
EUR 476.4

VPREB3 Polyclonal Antibody

A61726 EpiGentek
  • EUR 684.66
  • EUR 117.70
  • EUR 302.50
  • EUR 423.50
  • 100 µg
  • 20 ul
  • 50 ul
  • 100 ul

Vpreb3 sgRNA CRISPR/Cas9 All-in-One Lentivector set (Rat)

K6154005 ABM 3 x 1.0 ug
EUR 451.2

VPREB1 3'UTR GFP Stable Cell Line

TU078274 ABM 1.0 ml
EUR 1672.8

Vpreb1 3'UTR GFP Stable Cell Line

TU172112 ABM 1.0 ml Ask for price

Vpreb2 3'UTR GFP Stable Cell Line

TU172113 ABM 1.0 ml Ask for price

Vpreb1 3'UTR GFP Stable Cell Line

TU273237 ABM 1.0 ml Ask for price

Vpreb2 3'UTR GFP Stable Cell Line

TU273238 ABM 1.0 ml Ask for price

Vpreb3 sgRNA CRISPR/Cas9 All-in-One Lentivector set (Mouse)

K3123205 ABM 3 x 1.0 ug
EUR 451.2

Vpreb3 sgRNA CRISPR/Cas9 All-in-One Lentivector (Rat) (Target 1)

K6154006 ABM 1.0 ug DNA
EUR 200.4

Vpreb3 sgRNA CRISPR/Cas9 All-in-One Lentivector (Rat) (Target 2)

K6154007 ABM 1.0 ug DNA
EUR 200.4

Vpreb3 sgRNA CRISPR/Cas9 All-in-One Lentivector (Rat) (Target 3)

K6154008 ABM 1.0 ug DNA
EUR 200.4

VPREB3 Antibody, HRP conjugated

1-CSB-PA891724LB01HU Cusabio
  • EUR 380.40
  • EUR 402.00
  • 100ug
  • 50ug
Description: A polyclonal antibody against VPREB3. Recognizes VPREB3 from Human. This antibody is HRP conjugated. Tested in the following application: ELISA

VPREB3 Recombinant Protein (Rat)

RP236981 ABM 100 ug Ask for price

VPREB3 Antibody, FITC conjugated

1-CSB-PA891724LC01HU Cusabio
  • EUR 380.40
  • EUR 402.00
  • 100ug
  • 50ug
Description: A polyclonal antibody against VPREB3. Recognizes VPREB3 from Human. This antibody is FITC conjugated. Tested in the following application: ELISA

Vpreb3 sgRNA CRISPR/Cas9 All-in-One Lentivector (Mouse) (Target 1)

K3123206 ABM 1.0 ug DNA
EUR 200.4

Vpreb3 sgRNA CRISPR/Cas9 All-in-One Lentivector (Mouse) (Target 2)

K3123207 ABM 1.0 ug DNA
EUR 200.4

Vpreb3 sgRNA CRISPR/Cas9 All-in-One Lentivector (Mouse) (Target 3)

K3123208 ABM 1.0 ug DNA
EUR 200.4

Vpreb3 ORF Vector (Rat) (pORF)

ORF078995 ABM 1.0 ug DNA
EUR 607.2

VPREB3 Recombinant Protein (Mouse)

RP184868 ABM 100 ug Ask for price

Vpreb1 3'UTR Luciferase Stable Cell Line

TU122112 ABM 1.0 ml Ask for price

Vpreb2 3'UTR Luciferase Stable Cell Line

TU122113 ABM 1.0 ml Ask for price

VPREB1 3'UTR Luciferase Stable Cell Line

TU028274 ABM 1.0 ml
EUR 1672.8

Vpreb1 3'UTR Luciferase Stable Cell Line

TU223237 ABM 1.0 ml Ask for price

Vpreb2 3'UTR Luciferase Stable Cell Line

TU223238 ABM 1.0 ml Ask for price

VPREB3 Antibody, Biotin conjugated

1-CSB-PA891724LD01HU Cusabio
  • EUR 380.40
  • EUR 402.00
  • 100ug
  • 50ug
Description: A polyclonal antibody against VPREB3. Recognizes VPREB3 from Human. This antibody is Biotin conjugated. Tested in the following application: ELISA

Vpreb3 ORF Vector (Mouse) (pORF)

ORF061624 ABM 1.0 ug DNA
EUR 607.2

Human Pre- B lymphocyte protein 3, VPREB3 ELISA KIT

ELI-51048h Lifescience Market 96 Tests
EUR 988.8

Human Pre-B-Lymphocyte Gene 3(VPREB3) ELISA Kit

QY-E01939 Qayee Biotechnology 96T
EUR 433.2

Gene Knock-Out HR Targeting Vector [MCS1-EF1α-RFP-T2A-Puro-pA-MCS2]

HR110PA-1 SBI 10 ug
EUR 933

Gene Knock-Out HR Targeting Vector [MCS1-EF1a-GFP-T2A-Puro-pA-MCS2]

HR410PA-1 SBI 10 ug
EUR 933

VPREB3 Polyclonal Antibody, HRP Conjugated

A61729 EpiGentek
  • EUR 684.66
  • EUR 302.50
  • EUR 423.50
  • 100 µg
  • 50 ul
  • 100 ul

Gene Knock-Out HR Targeting Vector [MCS1-EF1a-RFP-T2A-Hygro-pA-MCS2]

HR510PA-1 SBI 10 ug
EUR 933

Polyclonal VPREB3 Antibody (internal region)

APG00623G Leading Biology 0.1mg
EUR 580.8
Description: A polyclonal antibody raised in Goat that recognizes and binds to Human VPREB3 (internal region). This antibody is tested and proven to work in the following applications:

VPREB3 Polyclonal Antibody, FITC Conjugated

A61728 EpiGentek
  • EUR 684.66
  • EUR 302.50
  • EUR 423.50
  • 100 µg
  • 50 ul
  • 100 ul

Pre-B Lymphocyte 3 (VPREB3) Antibody

20-abx114658 Abbexa
  • EUR 878.40
  • EUR 477.60
  • 150 ul
  • 50 ul

VPREB3 Polyclonal Antibody, Biotin Conjugated

A61727 EpiGentek
  • EUR 684.66
  • EUR 302.50
  • EUR 423.50
  • 100 µg
  • 50 ul
  • 100 ul

B2M Knockout Jurkat Cell Line

78342 BPS Bioscience 2 vials
EUR 6500
Description: B2M (Beta-2-Microglobulin) has been genetically removed by CRISPR/Cas9 genome editing from Jurkat cells.

TCR Knockout Jurkat Cell Line

78539 BPS Bioscience 2 vials
EUR 6500
Description: The TCR Knockout Jurkat cell line was generated by CRISPR/Cas9 genome editing to remove the TRAC (T-Cell Receptor Alpha Constant) and TRBC1 (T-Cell Receptor Beta Constant 1) domains of the TCRα and β chains.

B2M Knockout THP-1 Cell Line

78389 BPS Bioscience 2 vials
EUR 6500
Description: B2M (Beta-2-Microglobulin) has been genetically removed by CRISPR/Cas9 genome editing from THP-1 cells.

VPREB3 Protein Vector (Rat) (pPM-C-HA)

PV315980 ABM 500 ng
EUR 723.6

VPREB3 Protein Vector (Rat) (pPB-C-His)

PV315978 ABM 500 ng
EUR 723.6

VPREB3 Protein Vector (Rat) (pPB-N-His)

PV315979 ABM 500 ng
EUR 723.6

VPREB3 Protein Vector (Rat) (pPM-C-His)

PV315981 ABM 500 ng
EUR 723.6

VPREB3 Protein Vector (Mouse) (pPM-C-HA)

PV246496 ABM 500 ng
EUR 723.6

CIITA Knockout THP-1 Cell Line

78390 BPS Bioscience 2 vials
EUR 6500
Description: CIITA (Class II Transactivator) has been genetically removed from THP-1 cells using CRISPR/Cas9 genome editing.

Basic HR Targeting Vector [MCS1-LoxP-MCS2-MCS3-pA-LoxP-MCS4] for Gene Knock-In/Out

HR100PA-1 SBI 10 ug
EUR 868

VPREB3 Protein Vector (Mouse) (pPB-C-His)

PV246494 ABM 500 ng
EUR 723.6

VPREB3 Protein Vector (Mouse) (pPB-N-His)

PV246495 ABM 500 ng
EUR 723.6

VPREB3 Protein Vector (Mouse) (pPM-C-His)

PV246497 ABM 500 ng
EUR 723.6

TCR/B2M Knockout Jurkat Cell Line

78552 BPS Bioscience 2 vials
EUR 8645
Description: This cell line is a double knockout of TCR (T Cell Receptor) and B2M (Beta-2-Microglobulin). First, the TRAC (T-Cell Receptor Alpha Constant) and the TRBC1 (T-Cell Receptor Beta Constant 1) domains of the TCRα/β chains were genetically removed by CRISPR/Cas9 genome editing from Jurkat cells to generate the TCR Knockout Jurkat cell Line (BPS Bioscience #78539). These TCR knockout cells were then used to generate a new cell line in which B2M was also genetically removed by CRISPR/Cas9 genome editing.

293AD Cell Line

AD-100 Cell Biolabs 1 vial
EUR 553.2
Description: The 293AD Cell Line is derived from the parental 293 cells but selected for attributes that increase adenovirus production, including firmer attachment and larger surface area.

293AAV Cell Line

AAV-100 Cell Biolabs 1 vial
EUR 609.6
Description: The 293AAV Cell Line is derived from the parental 293 cells but selected for attributes that increase AAV production, including firmer attachment and larger surface area.

293LTV Cell Line

LTV-100 Cell Biolabs 1 vial
EUR 609.6
Description: The 293LTV Cell Line is derived from the parental 293 cells but selected for attributes that increase lentiviral production, including fimrer attachment and larger surface area.

293RTV Cell Line

RV-100 Cell Biolabs 1 vial
EUR 609.6
Description: The 293RTV Cell Line is derived from the parental 293 cells but selected for attributes that increase retroviral production, including fimrer attachment and larger surface area.
×

Szybka izolacja antygenów z komórek z adsorbentem gronkowcowym białko A-przeciwciało: parametry interakcji kompleksów przeciwciało-antygen z białkiem A.

The Cowan I pressure of the bacterium Staphylococcus aureus has been used as an adsorbent for antibodies complexed with radiolabeled antigens from cell lysates. This software is superior as a superior various to different strategies of immune precipitation for the isolation of antigens. It exploits the excessive adsorption capability for IgG molecules by protein A molecules on the cell partitions of sure strains of staphylococci, together with the advantageous sedimentation properties of the micro organism. The interplay of immune complexes with the adsorbent was outlined initially utilizing a mannequin system of bovine serum albumin with a excessive extra of rabbit anti-bovine serum albumin antibodies (IgG).

The uptake of immune complexes below these circumstances was extraordinarily speedy, occurring inside seconds, whereas most binding of free IgG was a lot slower. As well as, as soon as sure the complexed antigen couldn’t be displaced from the adsorbent both by giant quantities of regular IgG or by further free antibody. Antigen might be eluted virtually fully from the inert adsorbent for analytic or preparative functions with a wide range of solvent techniques, such because the detergent SDS in mixture with urea and excessive temperature, and impartial salts with robust lyotropic salting in properties.

The efficacy of the protein A-antibody adsorption method was examined in direct comparisons with a traditional double antibody precipitation methodology for the isolation of mouse lymphocyte IgM. The bacterial adsorbent not solely had a definite benefit in velocity of antigen isolation, however analyses by polyacrylamide gel electrophoresis in SDS additionally revealed constantly larger antigen recoveries, decrease ranges of background radioactivity, and an absence of different cell parts which can nonspecifically bind to and complicate analyses utilizing typical immune precipitates.

Antygen CD4 (T4) jest podstawowym składnikiem receptora retrowirusa AIDS.

Acquired immune deficiency syndrome (AIDS) is characterised by opportunistic infections and by ‘opportunistic neoplasms’ (for instance, Kaposi’s sarcoma). Persistent generalized lymphadenopathy (PGL) is epidemiologically related to AIDS, particularly in male homosexuals. A subset of T lymphocytes constructive for the CD4 antigen (additionally termed T4 antigen), is depleted in AIDS and PGL sufferers. A retrovirus present in T-cell cultures from these sufferers is strongly implicated within the aetiology of AIDS due to the excessive frequency of isolation and the prevalence of particular antibodies within the sufferers. Right here we’ve got detected cell-surface receptors for the AIDS retrovirus (human T-cell leukaemia virus-III (HTLV-III) and lymphadenopathy-associated virus-1 (LAV-1) isolates) by testing the susceptibility of cells to an infection with pseudotypes of vesicular stomatitis virus bearing retroviral envelope antigens, and by the formation of multinucleated syncytia on mixing virus-producing cells with receptor-bearing cells.

Receptors have been current solely on cells expressing CD4 antigen; amongst 155 monoclonal antibodies examined, every of the 14 anti-CD4 antibodies inhibited formation of syncytia and blocked pseudotypes. Productive an infection of CD4+ cells with HTLV-III or LAV-1 markedly decreased cell-surface expression of CD4. In distinction, receptors for HTLV-I and HTLV-II weren’t restricted to CD4+ cells, weren’t blocked by anti-CD4 antibodies; cells productively contaminated with HTLV-I and HTLV-II expressed floor CD4. Therefore, we conclude that the CD4 antigen is a vital and particular part of the receptor for the causative agent of AIDS.

Komórkowe i molekularne mechanizmy zwłóknienia.

Fibrosis is outlined by the overgrowth, hardening, and/or scarring of assorted tissues and is attributed to extra deposition of furthercellular matrix parts together with collagen. Fibrosis is the tip results of persistent inflammatory reactions induced by a wide range of stimuli together with persistent infections, autoimmune reactions, allergic responses, chemical insults, radiation, and tissue damage. Though present remedies for fibrotic ailments reminiscent of idiopathic pulmonary fibrosis, liver cirrhosis, systemic sclerosis, progressive kidney illness, and cardiovascular fibrosis sometimes goal the inflammatory response, there may be accumulating proof that the mechanisms driving fibrogenesis are distinct from these regulating irritation. In actual fact, some research have steered that ongoing irritation is required to reverse established and progressive fibrosis.

The important thing cellular mediator of fibrosis is the myofibroblast, which when activated serves as the first collagen-producing cell. Myofibroblasts are generated from a wide range of sources together with resident mesenchymal cells, epithelial and endothelial cells in processes termed epithelial/endothelial-mesenchymal (EMT/EndMT) transition, in addition to from circulating fibroblast-like cells referred to as fibrocytes which might be derived from bone-marrow stem cells. Myofibroblasts are activated by a wide range of mechanisms, together with paracrine indicators derived from lymphocytes and macrophages, autocrine elements secreted by myofibroblasts, and pathogen-associated molecular patterns (PAMPS) produced by pathogenic organisms that work together with sample recognition receptors (i.e. TLRs) on fibroblasts. Cytokines (IL-13, IL-21, TGF-beta1), chemokines (MCP-1, MIP-1beta), angiogenic elements (VEGF), progress elements (PDGF), peroxisome proliferator-activated receptors (PPARs), acute section proteins (SAP), caspases, and parts of the renin-angiotensin-aldosterone system (ANG II) have been recognized as essential regulators of fibrosis and are being investigated as potential targets of antifibrotic medicine. This evaluate explores our present understanding of the cellular and molecular mechanisms of fibrogenesis.

Predykcyjne korelaty odpowiedzi na przeciwciało anty-PD-L1 MPDL3280A u pacjentów z rakiem.

The event of human most cancers is a multistep course of characterised by the buildup of genetic and epigenetic alterations that drive or replicate tumour development. These adjustments distinguish most cancers cells from their regular counterparts, permitting tumours to be acknowledged as international by the immune system. Nevertheless, tumours are not often rejected spontaneously, reflecting their capability to keep up an immunosuppressive microenvironment. Programmed death-ligand 1 (PD-L1; additionally referred to as B7-H1 or CD274), which is expressed on many most cancers and immune cells, performs an essential half in blocking the ‘most cancers immunity cycle’ by binding programmed death-1 (PD-1) and B7.1 (CD80), each of that are destructive regulators of T-lymphocyte activation. Binding of PD-L1 to its receptors suppresses T-cell migration, proliferation and secretion of cytotoxic mediators, and restricts tumour cell killing.

The PD-L1-PD-1 axis protects the host from overactive T-effector cells not solely in most cancers but additionally throughout microbial infections. Blocking PD-L1 ought to due to this fact improve anticancer immunity, however little is thought about predictive elements of efficacy. This examine was designed to guage the protection, exercise and biomarkers of PD-L1 inhibition utilizing the engineered humanized antibody MPDL3280A. Right here we present that throughout a number of most cancers varieties, responses (as evaluated by Response Analysis Standards in Stable Tumours, model 1.1) have been noticed in sufferers with tumours expressing excessive ranges of PD-L1, particularly when PD-L1 was expressed by tumour-infiltrating immune cells. Moreover, responses have been related to T-helper kind 1 (TH1) gene expression, CTLA4 expression and the absence of fractalkine (CX3CL1) in baseline tumour specimens. Collectively, these knowledge counsel that MPDL3280A is best in sufferers by which pre-existing immunity is suppressed by PD-L1, and is re-invigorated on antibody remedy.

Dodaj komentarz Anuluj pisanie odpowiedzi

Twój adres e-mail nie zostanie opublikowany. Wymagane pola są oznaczone *

Ostatnie wpisy

  • Przygotowanie silnika do hamowania działań
  • Ryboflawina jako obiecujący środek przeciwdrobnoustrojowy
  • Analiza immunohistochemiczna ekspresji MCT1, MCT2 i MCT4 w mięśniu podeszwowym szczura.
  • Wieloośrodkowe, randomizowane badanie z podwójnie ślepą próbą
  • Niedobór alfa1-antytrypsyny

Najnowsze komentarze

    Kategorie

    • exosome
    • immune
    • Isotype
    • miRNA
    • PCR
    • peptide
    • Polyclonal
    • Purification
    • Real-time
    • Recombinant
    • Uncategorized

    Archiwa

    • czerwiec 2022
    • czerwiec 2021
    • maj 2021

    Kategorie

    • exosome
    • immune
    • Isotype
    • miRNA
    • PCR
    • peptide
    • Polyclonal
    • Purification
    • Real-time
    • Recombinant
    • Uncategorized

    Meta

    • Zaloguj się
    • Kanał wpisów
    • Kanał komentarzy
    • WordPress.org
    wrzesień 2023
    P W Ś C P S N
     123
    45678910
    11121314151617
    18192021222324
    252627282930  
    « cze    

    Beads and fluorescent Particles

    Kategorie

    • exosome
    • immune
    • Isotype
    • miRNA
    • PCR
    • peptide
    • Polyclonal
    • Purification
    • Real-time
    • Recombinant
    • Uncategorized

    Ostatnie wpisy

    • Przygotowanie silnika do hamowania działań
    • Ryboflawina jako obiecujący środek przeciwdrobnoustrojowy
    • Analiza immunohistochemiczna ekspresji MCT1, MCT2 i MCT4 w mięśniu podeszwowym szczura.
    • Wieloośrodkowe, randomizowane badanie z podwójnie ślepą próbą
    • Niedobór alfa1-antytrypsyny

    Quick Links

    • Contact Us
    • Distributors
    • Home
    • Konferencje
    • Nagrody i Stypendia
    • Projekty Badawcze
    • Publikacje
    © 2023 Hoxyl beads | Powered by Superbs Personal Blog theme